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Another important form of NO-related posttranslational modification (PTM) is protein S-nitrosylation (SNO), a product of the interaction of NO with the sulfhydryl groups of cysteine which leads to the formation of S-nitrosothiols [147, 159]

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Approaches that suppress lactate production, such as using PDK inhibitors (DCA and thiamine), have been demonstrated to alleviate organ damage and mitigate ferroptosis in sepsis by reversing the pathological Warburg effect-like state, thereby improving mitochondrial function and reducing ROS accumulation (189,190)

3d), whereas its overexpression (Extended Data Fig