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Pharmacologic activation of AMPK (e.g., by metformin or berberine) was shown to delay senescence and restore metabolic homeostasis, supporting its therapeutic potential in combating vascular aging and shear stress-related endothelial dysfunction [219]

Notably, inflammatory conditions may lead to a positive feedback loop in microglia, resulting in the secretion of proinflammatory mediators (Kuno et al., 2005), which can be neurotoxic and worsen existing damage (Zhang et al., 2004

This effect is mediated by promoting the inactivation of HMGB1, thereby effectively alleviating BLM-induced PF

Statements Author contributions LH: Conceptualization, Methodology, Writing original draft, Writing review & editing, Investigation, Software, Supervision, Validation, Visualization