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Significant increases in macrophages and neutrophils [68], pro-inflammatory cytokines (TNF- and IL-1) and chemokines (CCL2 and CCL3) [69], cell apoptosis [4], oxidative stress (reactive oxygen species and reactive nitrogen species), and the suppression of antioxidant proteins (such as glutathione peroxidase, catalase, and superoxide dismutase) are all frequently linked to COPD-induced airway obstruction [70] in both the airways and lung parenchyma, in response to noxious particles and gases [71]

The highest SE value observed in the CAA was 1.41 at the Ac-G combined dose of (1 + 0.125) g/mL, while the highest SE value observed in the cytoprotective effects was 1.61 at the Ac-G combined dose of (1 + 0.5) g/mL

[DOI] [PubMed] [Google Scholar] 35.Woo K.Y., Abbott L.K., Librach L

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