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241 Because skeletal muscle is the key site of insulin-dependent glucose disposal, the iron homeostasis on glycemia might be vitally important

Here we determined the mechanisms and rate constants for the glutathione/glutaredoxin-dependent redox reactions of the widely used redox probe roGFP2 and addressed the question of how mono- and dithiol glutaredoxins catalyze the reduction of roGFP2(S 2 ) as a model non-glutathione protein disulfide substrate

Presenilin mutations disrupt selenium uptake and thus inhibit GPX4 expression, increasing the sensitivity of multiple cell types to ferroptosis

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