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An alternative explanation is that inhibition of TS by folate-based agents is not durable, in spite of intracellular polyglutamation, and can be competed out by naturally occurring folates once plasma drug levels are reduced

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IL-23-induced synovial inflammation is primarily linked to the activation of JAK-STAT, tyrosine kinase 2, NF-B, and retinoic acid receptor-related orphan receptors (RORs) (80)

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