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Further experiments have demonstrated that the peptide is capable of suppressing the activity of pro-inflammatory and fibrotic factors characteristic of the senescence-associated secretory phenotype (SASP), thereby contributing to the restoration of damaged tissue microenvironments

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TB-500 Cellular Mechanisms and Receptor Interactions Actin-Binding Domain Functionality TB-500 functions primarily through high-affinity interactions with G-actin monomers, preventing spontaneous polymerisation and maintaining cytoplasmic actin pools in monomeric form